COVID-19 & Glutathione
There are many studies in which glutathione has been shown to have a positive effect on many other viral diseases and plays an important role in the development of these infections. Most viruses studied thrive in a low glutathione environment and high oxidative stress.
A recent study (see below) from China shows that moderate to severe Covid-19 patients are not able to make CD4+T and CD8+ T cells. These are the killer cells a persons immune system uses to kill viral and bacterial infections. This is why Glutathione is so critical. It allows the body to make CD4+T and CD8+ T cells to any level needed as long as you fuel Glutathione production.
Nrf2 expression modifies influenza A entry and replication in nasal epithelial cells
Ascorbic Acid Role in Containment of the World Avian Flu Pandemic
Drugs to cure avian influenza infection – multiple ways to prevent cell death
The cytokine storm of severe influenza and development of immunomodulatory therapy
Targeting Potential Drivers of COVID-19: Neutrophil Extracellular Traps
The Cellular Redox Environment Alters Antigen Presentation
Oxidative Stress During Viral Infection: A Review
RNA Viruses: ROS-Mediated Cell Death
Inhibition of Influenza Infection by Glutathione
Selenium, Selenoproteins and Viral Infection
Glutathione and HIV Infection: Reduced Reduced, or Increased Oxidized?
Glutathione and Immune Function
Oxidative Stress and Inflamatory Plasma Biomarkers in Respiratory Syncytial Virus Bronchiolitis
Oxidative Stress Modulation in Hepatitis C Virus Infected Cells
GSH and Analogs in Antiviral Therapy
Respiratory Syncytial Virus Induces Oxidative Stress by Modulating Antioxidant Enzymes
Antiviral and Immunomodulatory Properties of New Pro-Glutathione (GSH) Molecules
Antioxidant Therapy as a Potential Approach to Severe Influenza-Associated Complications
Clinical and immunological features of severe and moderate coronavirus disease 2019